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Compromised intestinal epithelial barrier due to intestinal epithelial-specific loss of JAM-A induces adaptive immune compensation that protects from colitis The findings in mice manipulated for MLCK or occludin expression differed from those obtained in mice lacking the JAM-A (encoded by F11r)
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Biotin, produced by intestinal bacteria, as well as that found in the diet, is bound to lysine residues in protein forming a complex that is called biocytin
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